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Tuesday, October 2, 2012

Beta-Blockers Might Not Reduce CV Events in Patients with Stable Heart Disease - Physician's First Watch

Beta-Blockers Might Not Reduce CV Events in Patients with Stable Heart Disease

Beta-blockers might not lower the risk for major cardiovascular events in patients with — or at risk for — stable coronary artery disease (CAD), according to a JAMA study.

Investigators compared outcomes with and without beta-blocker therapy in about 22,000 participants in the REACH (Reduction of Atherothrombosis for Continued Health) registry who had prior myocardial infarction, CAD without MI, or CAD risk factors only. Patients were followed for roughly 44 months.

In both cohorts with CAD, risk for the primary outcome — a composite of cardiovascular death, MI, or stroke — did not differ significantly between beta-blocker recipients and nonrecipients. In the risk-factor-only group, the primary outcome occurred more often among beta-blocker recipients (14% vs. 12% among nonrecipients).

The authors note that their findings support recent changes in secondary prevention guidelines, giving class I status only to the short-term use of beta-blockers after MI.

LINK(S):

JAMA article (Free abstract)

Journal Watch Cardiology summary (Free)

Background: Journal Watch Cardiology coverage of recent secondary prevention guidelines (Free)

Published in Physician's First Watch October 3, 2012



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Fish linked to heart failure risk, omega-3 results mixed

Fish linked to heart failure risk, omega-3 results mixed

By Andrew M. Seaman

NEW YORK (Reuters Health) - For people conscious about their heart health, a new study suggests it may be best to eat fish instead of taking individual omega-3 fatty acids in supplement form.

After reviewing information on the habits and fatty-acid blood levels of more than 20,000 male doctors, researchers found mixed results when it came to omega-3 supplements and the men's likelihood of heart failure, but eating fish regularly was linked to a lower risk.

According to the study's senior researcher, the results are consistent with the American Heart Association's (AHA) recommendations.

"Our findings showed a lower risk of heart failure in men consuming any amount of fish per week," Dr. Luc Djoussé, a professor at Harvard Medical School in Boston said in an email. "Given current AHA recommendations, we do not believe that any change should be made based on our findings."

The current recommendations are for people to eat two servings of fatty fish per week. Salmon, herring, sardines and albacore tuna are some of the fish considered the most beneficial, according to the AHA.

Some people, however, may prefer to take omega-3 fatty acid supplements that are available over the counter, but their benefits for heart health remain unclear.

One recent study, for instance, analyzed data from multiple past studies and found no link between omega-3 supplements and overall death rates (see Reuters Health story from September 11, 2012).

For the new study, published in the American Journal of Clinical Nutrition, Djoussé's group wanted to see if there was any connection between taking individual omega-3 fatty acids and one's risk of heart failure.

They used information from the Physicians' Health Study, an ongoing study of male doctors that began in 1982.

The researchers analyzed how much omega-3 fatty acid the doctors had in their bodies based on blood samples and food questionnaires, which also indicated how much fish the doctors ate.

They found that about 7 in 1,000 men who reported eating fish less than once per month suffered heart failure. That's compared to about 4 in 1,000 men who reported eating more than one serving of fish per month.

Djoussé said that works out to be "about 30 percent lower risk of heart failure than that of men not consuming fish."

DHA, EPA, DPA

As for the individual omega-3 fatty acids, things were a little more complicated.

Specifically, the researchers looked at eicosapentaenoic acid (EPA), docosahexaenoic acid (DHA) and docosapentaenoic acid (DPA) in the men's diets and bloodstreams.

Overall, the researchers did not see an effect for blood levels of EPA or DHA, which are the two fatty acids thought to be responsible for fish's benefits and most often sold in the form of "fish oil" supplements.

There was a link between lowered heart failure risk and DPA, which is made in the body from EPA and can then be converted into DHA, according to Djoussé.

He told Reuters Health that their findings suggest DPA acts differently than the other fatty acids, but that needs to be confirmed by more research.

Also, while the researchers did see a link between eating fish and a lower heart failure risk, the study cannot prove whether or not the fish caused the lower risk.

Alice Lichtenstein, director of the Cardiovascular Nutrition Laboratory at Tufts University in Boston, told Reuters Health the study shows that the hope for a "quick fix" is fading.

"What we need to do - and I think this maybe reinforces it - is to put more effort on modifying the diet of people who are at an increased risk," she said.

For example, eating fish may not be just beneficial thanks to the omega-3 fatty acids, but also because it displaces less healthy foods from a person's diet.

Djoussé said until more studies are done, people should not change their diets as long as they're already following the AHA's guidelines.

SOURCE: http://bit.ly/UDVpOA American Journal of Clinical Nutrition, online September 5, 2012.



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Friday, September 21, 2012

Eating yogurt may prevent high blood pressure

Eating Yogurt May Protect Against High Blood Pressure

21 Sep 2012   

A new study has suggested that adding low calorie yogurt to your diet may help lower your risk of high blood pressure.

The new information presented at the American Heart Association's High Blood Pressure Research 2012 Scientific Sessions, has found that long-term yogurt eaters had a lower systolic blood pressure, as well as a diminished risk of developing high blood pressure.

Systolic blood pressure is the measure of how powerful the blood is against the walls of your arteries when your heart is beating. It is the top number in a blood pressure reading.

Hypertension can be a commonly misunderstood condition. According to the American Heart Association,untreated high blood pressure damages and scars your arteries. Tears and scars in the arteries can act like a net catching debris traveling through the bloodstream, such as cholesterol and plaque, which can then lead to build ups or blockages. It is important to be aware of blood pressure measurements, because symptoms are many times non-existent.

Past studies have established various health benefits of yogurt. Frequent consumption has been associated with healthier body weight and lower body mass index.

Low fat yoghurts with berries
Adding a low calorie yogurt to your daily diet may reduce your risk of high blood pressure.
Yogurt contains calcium, many needed daily nutrients and is easy to add to a meal or have as a snack. A great source of protein, yogurt keeps you feeling full slightly longer and also has liquid that provides hydration.

These researchers conducted a study lasting 15 years following more than 2,000 volunteers who did not have high blood pressure at the beginning of the study. Over the study period, participants filled out questionnaires three different times to measure yogurt intake.

Results showed that 31 percent of volunteers were less likely to develop high blood pressure when at least 2 percent of their daily caloric intake was yogurt, equivalent to at least one six-ounce cup of low-fat yogurt every three days. Their systolic blood pressure also increased significantly less than those who did not eat yogurt. These findings support a common belief that low-fat dairy products reduce blood pressure.

A healthy diet including low-fat yogurt, paired with physical activity, can help prevent chronic diseases such as hypertension and manage your health.

It should be noted that this study was presented at a conference and published as an abstract. It has not yet been published in a peer-reviewed journal.

Written by Kelly Fitzgerald
Copyright: Medical News Today
Not to be reproduced without permission of Medical News Today


References:
American Heart Association


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Sugary drinks and obesity: the link is real

Studies strengthen the case against sugary drinks as culprits in obesity epidemic

By Associated Press,  Published: FRIDAY, SEPTEMBER 21, 6:01 PM ET

  Aa  

New research powerfully strengthens the case against soda and other sugary drinks as culprits in the obesity epidemic.

A huge, decades-long study involving more than 33,000 Americans has yielded the first clear proof that drinking sugary beverages interacts with genes that affect weight, amplifying a person's risk of obesity beyond what it would be from heredity alone.

This means that such drinks are especially harmful to people with genes that predispose them to weight gain. And most of us have at least some of these genes.

In addition, two other major experiments have found that giving children and teens calorie-free alternatives to the sugary drinks they usually consume leads to less weight gain.

Collectively, the results strongly suggest that sugary drinks cause people to pack on the pounds, independent of other unhealthy behavior such as overeating and getting too little exercise, scientists say.

That adds weight to the push for taxes, portion limits like the one just adopted in New York City, and other policies to curb consumption of soda, juice drinks and sports beverages sweetened with sugar.

Soda lovers do get some good news: Sugar-free drinks did not raise the risk of obesity in these studies.

"You may be able to fool the taste" and satisfy a sweet tooth without paying a price in weight, said an obesity researcher with no role in the studies, Rudy Leibel of Columbia University.

The studies were being presented Friday at an obesity conference in San Antonio and were published online by the New England Journal of Medicine.

The gene research in particular fills a major gap in what we know about obesity. It was a huge undertaking, involving three long-running studies that separately and collectively reached the same conclusions. It shows how behavior combines with heredity to affect how fat we become.

Having many of these genes does not guarantee people will become obese, but if they drink a lot of sugary beverages, "they fulfill that fate," said an expert with no role in the research, Jules Hirsch of Rockefeller University in New York. "The sweet drinking and the fatness are going together, and it's more evident in the genetic predisposition people."

Sugary drinks are the single biggest source of calories in the American diet, and they are increasingly blamed for the fact that a third of U.S. children and teens and more than two-thirds of adults are obese or overweight.

Consumption of sugary drinks and obesity rates have risen in tandem — both have more than doubled since the 1970s in the U.S.

But that doesn't prove that these drinks cause obesity. Genes, inactivity and eating fatty foods or just too much food also play a role. Also, diet research on children is especially tough because kids are growing and naturally gaining weight.

Until now, high-quality experiments have not conclusively shown that reducing sugary beverages would lower weight or body fat, said David Allison, a biostatistician who has done beverage research at the University of Alabama at Birmingham, some of it with industry support.

He said the new studies on children changed his mind and convinced him that limiting sweet drinks can make a difference.

In one study, researchers randomly assigned 224 overweight or obese high schoolers in the Boston area to receive shipments every two weeks of either the sugary drinks they usually consumed or sugar-free alternatives, including bottled water. No efforts were made to change the youngsters' exercise habits or give nutrition advice, and the kids knew what type of beverages they were getting.

After one year, the sugar-free group weighed more than 4 pounds less on average than those who kept drinking sugary beverages.

"I know of no other single food product whose elimination can produce this degree of weight change," said the study's leader, Dr. David Ludwig of Boston Children's Hospital and the Harvard School of Public Health.

The weight difference between the two groups narrowed to 2 pounds in the second year of the study, when drinks were no longer being provided. That showed at least some lasting beneficial effect on kids' habits. The study was funded mostly by government grants.

A second study involved 641 normal-weight children ages 4 to 12 in the Netherlands who regularly drank sugar-sweetened beverages. They were randomly assigned to get either a sugary drink or a sugar-free one during morning break at their schools, and were not told what kind they were given.

After 18 months, the sugary-drink group weighed 2 pounds more on average than the other group.

The studies "provide strong impetus" for policies urged by the Institute of Medicine, the American Heart Association and others to limit sugary drink consumption, Dr. Sonia Caprino of the Yale School of Medicine wrote in an editorial in the journal.

The American Beverage Association disagreed.

"Obesity is not uniquely caused by any single food or beverage," it said in a statement. "Studies and opinion pieces that focus solely on sugar-sweetened beverages, or any other single source of calories, do nothing meaningful to help address this serious issue."

The genetic research was part of a much larger set of health studies that have gone on for decades across the U.S., led by the Harvard School of Public Health.

Researchers checked for 32 gene variants that have previously been tied to weight. Because we inherit two copies of each gene, everyone has 64 opportunities for these risk genes. The study participants had 29 on average.

Every four years, these people answered detailed surveys about their eating and drinking habits as well as things like smoking and exercise. Researchers analyzed these over several decades.

A clear pattern emerged: The more sugary drinks someone consumed, the greater the impact of the genes on the person's weight and risk of becoming obese.

For every 10 risk genes someone had, the risk of obesity rose in proportion to how many sweet drinks the person regularly consumed. Overall calorie intake and lifestyle factors such as exercise did not account for the differences researchers saw.

This means that people with genes that predispose them to be obese are more susceptible to the harmful effects of sugary drinks on their weight, said one of the study leaders, Harvard's Dr. Frank Hu. The opposite also was true — avoiding these drinks can minimize the effect of obesity genes.

"Two bad things can act together and their combined effects are even greater than either effect alone," Hu said. "The flip side of this is everyone has some genetic risk of obesity, but the genetic effects can be offset by healthier beverage choices. It's certainly not our destiny" to be fat, even if we carry genes that raise this risk.

The study was funded mostly by federal grants, with support from two drug companies for the genetic analysis.

___

Online:

Obesity info: http://www.cdc.gov/obesity/data/trends.html

BMI calculator: http://www.nhlbisupport.com/bmi/bminojs.htm

New England Journal: http://www.nejm.org

___

Marilynn Marchione can be followed at http://twitter.com/MMarchioneAP

Copyright 2012 The Associated Press. All rights reserved. This material may not be published, broadcast, rewritten or redistributed.



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Tuesday, September 18, 2012

Gastric bypass reduced cardiovascular, metabolic risks in severely obese | Cardiology

Gastric bypass reduced cardiovascular, metabolic risks in severely obese

Severely obese patients who underwent gastric bypass surgery had greater diabetes remission rates and were less likely to experience cardiovascular events than controls in a recent study.

In a prospective study, researchers evaluated 1,156 patients with a BMI of 35 or more who underwent Roux-en-Y gastric bypass surgery (n=418) (RYGB), sought weight loss surgery but did not undergo it (n=417, control group 1) or did not seek surgery (n=321, control group 2) between July 2000 and June 2011 in Utah.

Among evaluable participants, those who underwent bypass lost 27.7% of their initial weight, compared with 0.2% of control group 1 and 0% of control group 2. More than 20% of lost weight was maintained by the RYGB group in 94% of cases after 2 years and in 76% after 6 years.

Incidence rates for diabetes were lower in the RYGB group (2% compared with 17% in control group 1, OR=0.11, 0.04-0.34, and 15% in control group 2, OR=0.21, 0.06-0.67). Remission rates for diabetes after 6 years also were significantly greater in the bypass group (62% compared with 8% in control group 1, OR=16.5, 4.7-57.6, and 6% in control group 2, OR=21.5, 5.4-85.6) (95% CI for all).

Hypertension remission rates also improved for RYGB patients compared with control groups (42% vs. 18% in control group 1, OR=2.9, 1.4-6.0 and with 9% in control group 2, OR=5.0, 2.1-11.9). Remission rates for low high-density lipoprotein cholesterol levels saw gains in more bypass patients (67% of patients compared with 34% in control group 1, OR=3.8, 2.0-7.2 and with 18% in control group 2, OR=6.2, 2.7-14.1) (95% CI for all).

"Our study reports significant weight loss and 6-year improvements in major cardiovascular and metabolic risk factors in patients receiving RYGB surgery compared with severely obese control participants," the researchers wrote. "In contrast, cardiovascular and metabolic status of several obese control participants generally worsened. These findings are important considering the rapid increase in total numbers of bariatric surgical operations performed in the United States and worldwide, and may have significant ramifications for the projected 31 million US individuals meeting criteria for bariatric surgery."

Disclosure: See the study for a full list of relevant disclosures.



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Heart Disease and Firefighters: How and Why? - Fire Engineering

Heart Disease and Firefighters: How and Why?

By John Hofman

It has been well documented that the #1 killer of firefighters is heart disease. Intervention strategies within a comprehensive health and wellness program have helped to reduce these numbers over the years, but there is still an alarming rate of firefighters who suffer from heart attacks each year.

Researchers at Iowa State University discovered that 86 percent of volunteer firefighters did not know their blood lipid and 47 percent did not know their blood pressure (1). Within the U.S. alone, more than 1 million people will suffer a heart attack within the next year, and it does not just affect the elderly. We are now seeing individuals as young as 21 suffer from heart disease. So why are firefighters at a more increased risk than the average population?

Simple: It's the job! But let's take a closer look at some of the things that lead to an increase in heart attacks.

Sleep plays an important role in helping our bodies to recover from stress, illness, and fatigue. If we do not get quality sleep, our resting metabolic rate will decrease, causing weight gain. A firefighter's "internal biological clock" is often disrupted throughout a shift, affecting the body's ability to regulate the sleep-wake system. Researchers at the Brigham and Women's hospital showed that prolonged sleep restriction with simultaneous circadian disruption decreased the individual's metabolic rate, thereby increasing over time the risk for diabetes. (2)

The University of Chicago went even further and showed there is a direct link between sleeping and an increased risk of stroke, heart attack, and congestive heart failure. (3) Their findings showed how an individual who sleeps more than eight hours and fewer than six hours had a significantly higher chance of experiencing chest pain or angina and coronary heart disease. Therefore, it is important to control the duration of restful sleep in a completely dark room to help reduce the chances of heart disease and other related illnesses.

Abdominal fat, also known as visceral fat, is generally associated with diabetes. However, there have been links to an increase of strokes through the hardening of the arteries. Increased visceral fat could lead to diabetes, which creates a change in blood vessels that reduce blood flow to the brain. Based on the findings at Iowa State University, 41 percent of volunteer firefighters were classified as obese and 35 percent as overweight. Therefore, it is recommended that a firefighter participate in physical activity and proper nutrition programs to reverse the side effects associated with belly fat.

Firefighters are often exposed to traumatic stress. Over a 30-year career, things tend to affect them both mentally and physically. According to researchers at the University of California - San Francisco, these exposures over a lifetime or career will boost inflammation in the body, even if they do not lead to post-traumatic stress disorder. (4) It was discovered that the greater the traumatic stress, the higher levels of inflammation within the body. Individuals with higher levels of inflammation within their body tend to have an increased risk of having a heart attack. Even if the firefighter adjusted to these traumatic events, the inflammation remained constant over a period of time. So the stress of the job can impact your health even if you don't have certain mental or physical symptoms. Intervention strategies to help combat stress, such as exercise, yoga, and other health-related activities, should be integrated at the start of a firefighter's career.

Firefighting itself is physically demanding and will eventually break the body down. According to a study performed at the Illinois Fire Service Institute (5), three hours of prolonged firefighting stiffens arteries and impairs heart function in young, healthy male firefighters. The same is seen within heavy powerlifters and ultra-marathon runners. This could affect those firefighters who do not value the importance of fitness and, therefore, exhibit several of the risk factors for cardiovascular disease, including being overweight and having elevated blood pressure and/or cholesterol.

Finally, your lungs. Breathing in toxic fumes and particles during overhaul, exhaust pollution from the apparatus bay, and exposures during most regular calls can adversely affect lung function. Lung function and obstructive airway diseases are strongly and independently associated with increased risk of heart failure. These results were not limited to smokers, but also to nonsmokers.

So what can you do?

1.  Get an annual physical with a complete blood panel. In addition to the normal blood work, ask your physician to include the following:

a.  C-Reactive Protein (CRP) is elevated in the blood when there is widespread inflammation somewhere in the body. The evidence now available indicates that inflammation and molecules such as CRP associated with inflammation may be as important as cholesterol in determining the development of atherosclerosis (hardening of the arteries) and heart disease.

i.   You are at low risk of developing cardiovascular disease if your high sensitivity (hs)-CRP level is lower than 1.0mg/L.

ii.  You are at average risk of developing cardiovascular disease if it is between 1.0 and 3.0 mg/L.

iii. You are at high risk for cardiovascular disease if your hs-CRP level is higher than 3.0 mg/L.

2.  Testosterone: Lower testosterone levels have been shown to be an independent risk factor for worse outcomes among men and women with heart failure. It also has been associated with decreased survival for men with coronary artery disease.

3.  forget about diets and low fat! eat a more healthful diet. There is no magic pill! Although there are some benefits to taking omega -3 (usually taken in a fish oil supplement), some studies have suggested otherwise. In an analysis of past studies, there was no difference in the number of heart attacks, strokes, or deaths among more than 20,000 people with heart disease who were randomly assigned to take either fish oil supplements or fish oil-free placebo pills. So eat better.

•         Eat more fruits and vegetables, which are high in antioxidants. Good heart health depends on open, flexible arteries that can deliver blood efficiently throughout the body. Dark chocolate and cocoa, as well as plant-based compounds found in red wine and green tea, are high in antioxidants, which help fight cell damage from free radicals in the bloodstream that can cause fatty plaque to build up on artery walls

•         eat more fiber: Aim for 35 to 40 grams of dietary fiber per day. Fiber helps to lower cholesterol by binding with it and pulling it out of the system.

•         spice it up: Numerous studies have shown that spices can help improve cardiovascular health. Cayenne pepper is known to strengthen the heart, arteries, and capillaries and to lower the cholesterol level. Garlic is known to help lower blood cholesterol, and ginger is a natural blood thinner and anti-inflammatory agent.

4.  Exercise, exercise, exercise. 30 minutes of aerobic activity is associated with a 70-percent reduction in heart attack risk over a year. Researchers from the Mayo Clinic analyzed its data and noticed that a brisk 10-minute walk a day results in a nearly 50-percent reduction in heart attacks versus doing nothing. (7)

5.  Stay happy; be optimistic. It could save your life. A Duke University study of 255 doctors from several years ago found that 14 percent of those rated above average for hostility based on a personality test had died 25 years later--most from heart disease--compared with 2 percent of those who tested below the average. (8)

References

(1)  Yoo, HL, Franke, WD. "Prevalence of cardiovascular disease risk factors in volunteer firefighters." Department of Kinesiology, Iowa State University. J Occup Environ Med. Aug 2009; 51(8):958-62.

(2)  O M Buxton, SW Cain, SP O'Connor, JH Porter, JF Duffy, W Wang, CA Czeisler, SA Shea. "Adverse Metabolic Consequences in Humans of Prolonged Sleep Restriction Combined with Circadian Disruption." Science Translational Medicine, 2012; 4 (129):

(3)  American College of Cardiology (2012, March 26). "Sleeping too much or too little can be bad for your heart." ScienceDaily. Retrieved April 21, 2012.

(4)  O'Donovan A, Neylan TC, Metzler T, Cohen BE. Lifetime exposure to traumatic psychological stress is associated with elevated inflammation in the Heart and Soul Study. Brain Behav Immun. 2012 May; 26(4):642-9. Epub 2012 Feb 15.

(5)  Fahs, Christopher A, Huimin Yan, Ranadive, Sushant, Rossow, et al. "Acute effects of firefighting on arterial stiffness and blood flow." Vasc Med; April 2011 16: 113-118

(6)  Grace Rattue. "Lower Lung Function and Airflow Obstruction Raise Heart Failure Risk." Medical News Today. MediLexicon, Intl., 26 Feb. 2012. Web. 21 Apr. 2012.

(7)  MayoClinic.com: Walking for Fitness: How to Trim Your Waistline, Improve Your Health.

(8)  Barefoot JC, Brummett BH, Williams RB, et al. "Recovery expectations and long-term prognosis of patients with coronary heart disease." Arch Intern Med; 2011; DOI:10.1001/archinternmed.2011.41

John HofmanJohn Hofman is the strength and conditioning coach for the Sacramento (CA) Fire Department, He oversees the Wellness Center; coordinates the department's medical and fitness assessments; develops recruit fitness training, pre-employment medical and fitness evaluations; and assists the department's 20 certified Peer Fitness Trainers. In addition, he is the strength and conditioning coach for the California Regional Fire Academy, Sierra Fire Technology Program, Rocklin Fire Department, and South Placer Fire District. He also consults with the Fire Agency Self-Insurance System of California.



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Monday, September 17, 2012

Cancer passes heart disease as top killer of Hispanics

Cancer passes heart disease as top killer of Hispanics

Cancer is the leading cause of death among U.S. Hispanics, surpassing heart disease, American Cancer Society researchers report.

And although heart disease remains the No. 1 killer of non-Hispanic whites and African Americans, cancer is expected to assume the top spot within 10 years as prevention and treatment reduce heart disease deaths. Overall, death rates for both illnesses declined between 2000 and 2009.

"The overall message is positive," said Dr. Paulo Pinheiro, an epidemiologist at the University of Nevada not involved with the report, told the Los Angeles Times.

Using the most recent data available, the researchers report that 29,935 Hispanics died of cancer in 2009 and 29,611 from heart disease. Among all Americans in 2009, 599,413 died from heart disease and 567,628 from cancer, the U.S. Centers for Disease Control and Prevention.

Compared with other whites, Hispanics have higher incidences of and mortality rates for cancers of the stomach, liver, uterus, cervix and gallbladder, the cancer society says. For all cancers and for the four most common -- breast, prostate, lung and bronchus and colorectum -- Hispanics have incidence and mortality rates that are lower than other whites.

Hispanic women contract and die from cervical cancer at rates 50% to 70% higher than non-Hispanic whites.

The researchers said that higher rates reflected "greater exposure to cancer-causing infectious agents, lower rates of screening for cervical cancer, and possibly genetic factors," and that Hispanics "are diagnosed at an advanced stage of disease more often than non-Hispanic whites for most cancer sites."

Lung cancer rates for Hispanics, however, are about 50% less than other whites, because they are less likely to smoke tobacco.

Here's how the researchers summed up the cancer differences:

Much of the difference in the cancer burden among U.S. Hispanics results from their unique profile in terms of age distribution, socioeconomic status, and immigration history. Just one in ten U.S. Hispanics is 55 years or older, the age group among whom the majority of cancers are diagnosed, compared with almost one in three non-Hispanics. In 2010, more than one in four (26.6%) Hispanics lived in poverty and nearly one in three (30.7%) was uninsured, compared with 9.9% and 11.7%, respectively, of non-Hispanic whites.

Hispanics in the U.S. are an extremely diverse group because they originate from many different countries (e.g., Mexico, Central and South America, and Cuba). As a result, cancer patterns among Hispanic subpopulations vary substantially. For example, in Florida the cancer death rate among Cuban men is double that of Mexican men.

The finding appear in CA: A Cancer Journal for Clinicians.

Datos y Estadísticas sobre el Cáncer entre los Hispanos/Latinos 2009-2011.